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Neonatal issues

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9 papers 100 to 500 followers
By Faye Kehler Family Physician and GP Anesthetist since 1987 interested in all aspects of Medicine
David H Adamkin
A consistent definition for neonatal hypoglycemia in the first 48 h of life continues to elude us. Enhanced understanding of metabolic disturbances and genetic disorders that underlie alterations in postnatal glucose homeostasis has added useful information to understanding transitional hypoglycemia. This growth in knowledge still has not led to what we need to know: "How low is too low and for how long?" This article reviews the current state of understanding of neonatal hypoglycemia and how different approaches reach different "expert" opinions...
February 2017: Seminars in Fetal & Neonatal Medicine
Kimberly Dow, Alice Ordean, Jodie Murphy-Oikonen, Jodie Pereira, Gideon Koren, Henry Roukema, Peter Selby, Ruth Turner
Ontario's clinical practice guidelines for neonatal abstinence syndrome (NAS) provide evidence-informed recommendations that address the needs of substance using pregnant women and newborns at risk of NAS. NAS is a complex and multifaceted issue that is escalating along with rapidly rising opioid use in Ontario. Reducing the incidence and impact of NAS requires immediate action in order to improve the care of affected women and infants. This includes optimizing and standardizing treatment strategies, assessing and managing social risk, better monitoring of prescribing practices and facilitating the implementation of better treatment and prevention strategies as they become available...
2012: Journal of Population Therapeutics and Clinical Pharmacology
D H Adamkin, R Polin
No abstract text is available yet for this article.
January 2016: Journal of Perinatology: Official Journal of the California Perinatal Association
Myra H Wyckoff, Khalid Aziz, Marilyn B Escobedo, Vishal S Kapadia, John Kattwinkel, Jeffrey M Perlman, Wendy M Simon, Gary M Weiner, Jeanette G Zaichkin
No abstract text is available yet for this article.
November 2015: Pediatrics
Courtney J Wusthoff, Irene M Loe
Bilirubin-induced neurologic dysfunction (BIND) is the constellation of neurologic sequelae following milder degrees of neonatal hyperbilirubinemia than are associated with kernicterus. Clinically, BIND may manifest after the neonatal period as developmental delay, cognitive impairment, disordered executive function, and behavioral and psychiatric disorders. However, there is controversy regarding the relative contribution of neonatal hyperbilirubinemia versus other risk factors to the development of later neurodevelopmental disorders in children with BIND...
February 2015: Seminars in Fetal & Neonatal Medicine
Yvonne W Wu, Michael W Kuzniewicz, Andrea C Wickremasinghe, Eileen M Walsh, Soora Wi, Charles E McCulloch, Thomas B Newman
IMPORTANCE: Exchange transfusion is recommended for newborns with total serum bilirubin (TSB) levels thought to place them at risk for cerebral palsy (CP). However, the excess risk for CP among these infants is unknown. OBJECTIVE: To quantify the risks for CP and CP consistent with kernicterus that are associated with high TSB levels based on the 2004 American Academy of Pediatrics exchange transfusion threshold (ETT) guidelines. DESIGN, SETTING, AND PARTICIPANTS: We enrolled 2 cohorts from a population of 525,409 infants in the Late Impact of Getting Hyperbilirubinemia or Phototherapy (LIGHT) birth cohort...
March 2015: JAMA Pediatrics
Ichiro Morioka, Sota Iwatani, Tsubasa Koda, Kazumoto Iijima, Hajime Nakamura
Bilirubin-induced neurologic dysfunction (BIND) is a syndrome of subtle bilirubin neurotoxic disorders. The risk for developing BIND in newborns usually increases with elevated serum/plasma concentrations of unconjugated bilirubin. This risk is further increased by disorders of bilirubin binding to albumin, which includes a reduction in serum albumin concentrations or in the bilirubin-binding capacity and affinity of albumin, and the presence of displacing substances or infection. Serum unbound bilirubin (UB) concentration may be an ideal marker that reflects changes in bilirubin binding to albumin...
February 2015: Seminars in Fetal & Neonatal Medicine
Win Tin
Hypoglycaemia is one of the most frequent metabolic problems in neonatal medicine, and maintaining glucose homeostasis is one of the important physiological events during fetal-to-neonatal transition. Although frequently observed transient low blood glucose concentrations in the majority of healthy newborns are the reflections of normal metabolic adaptation processes during this transition, there has been a genuine concern that prolonged or recurrent low blood glucose levels may result in acute systemic effects and long-term neurological and developmental consequences...
February 2014: Seminars in Fetal & Neonatal Medicine
Roelineke J Lunsing
Subtle bilirubin-induced neurological dysfunction (BIND) is defined as disturbances in sensory and sensorimotor integration, central auditory processing, coordination, and muscle tone in the absence of the classical findings of kernicterus. This review is restricted to the (sensori)motor signs of BIND associated with unconjugated hyperbilirubinemia in term and late preterm neonates. The diagnosis of BIND at follow-up requires validated, age-specific techniques that are designed to identify these disturbances in infancy and later childhood...
November 2014: Seminars in Perinatology
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