We have located links that may give you full text access.
ENGLISH ABSTRACT
JOURNAL ARTICLE
REVIEW
[Pathogenesis of erythrocyte destruction in autoimmune hemolytic anemia].
Nihon Rinsho. Japanese Journal of Clinical Medicine 1996 September
The pathogenesis of autoimmune hemolytic anemias, including warm type autoimmune hemolytic anemia (AIHA), cold agglutinin disease (CAD), and paroxysmal cold hemoglobinuria (PCH), is described. These hemolytic anemias are characterized by shortening of erythrocyte survival in vivo and evidence of host antibodies reactive with autologous erythrocytes. Although the etiology of erythrocyte autoantibodies remains to be solved, it is established that they are pathogenic. Warm-reactive antibody usually facilitates the sequestration of sensitized erythrocytes by macrophages in spleen and liver, whereas cold-reactive antibodies cause either immediate intravascular hemolysis of sensitized erythrocytes by complement-mediated mechanism or their sequestration by macrophages.
Full text links
Trending Papers
The ten commandments of point-of-care ultrasound (POCUS).CJEM 2023 November 17
Restrictive or Liberal Transfusion Strategy in Myocardial Infarction and Anemia.New England Journal of Medicine 2023 November 12
Cushing's syndrome.Lancet 2023 November 18
Get seemless 1-tap access through your institution/university
For the best experience, use the Read mobile app
Read by QxMD is copyright © 2021 QxMD Software Inc. All rights reserved. By using this service, you agree to our terms of use and privacy policy.
You can now claim free CME credits for this literature searchClaim now
Get seemless 1-tap access through your institution/university
For the best experience, use the Read mobile app