Add like
Add dislike
Add to saved papers

Laxity of the ulnar nerve during elbow flexion and extension.

PURPOSE: To evaluate the dynamic anatomy of the ulnar nerve at the elbow.

METHODS: We studied 11 fresh cadavers. We placed metal clips on the ulnar nerve at three locations: at the medial epicondyle (point A), 3 cm proximal to the epicondyle (point B), and 14 cm proximal to the epicondyle (point C). The distances from the medial epicondyle to points A, B, and C on the ulnar nerve and between each pair of points were measured in full elbow extension and flexion.

RESULTS: With full elbow flexion, there was no movement of the ulnar nerve at point A (adjacent to the medial epicondyle). Point A and the adjacent distal ulnar nerve moved as a unit with the forearm around the medial epicondyle. Proximal to the cubital tunnel, there was significant ulnar nerve excursion (P < .01) at points B (0.7 ± 0.3 cm) and C (0.2 ± 0.2 cm). There was differential excursion of the ulnar nerve at points B and C relative to the medial epicondyle. The distances between the markers revealed that the nerve did not stretch to account for the discrepant distances of the 3 points, but a slack region of the nerve proximal to the medial epicondyle was taken up with flexion. Release of the intermuscular septum and the canal of Struthers did not influence movement of the nerve.

CONCLUSIONS: With elbow flexion, the ulnar nerve did not move appreciably in the distal-proximal direction directly at the cubital tunnel, but maximal excursion was in the fatty region proximal to the elbow. This slack region of the nerve was taken up during flexion, whereas only 2 mm of motion occurred through the canal of Struthers. The slack region might predispose to subluxation of the nerve. Conversely, decreased laxity might result in increased traction of the nerve, contributing to cubital tunnel syndrome.


Full text links

We have located links that may give you full text access.
Can't access the paper?
Try logging in through your university/institutional subscription. For a smoother one-click institutional access experience, please use our mobile app.

For the best experience, use the Read mobile app

Group 7SearchHeart failure treatmentPapersTopicsCollectionsEffects of Sodium-Glucose Cotransporter 2 Inhibitors for the Treatment of Patients With Heart Failure Importance: Only 1 class of glucose-lowering agents-sodium-glucose cotransporter 2 (SGLT2) inhibitors-has been reported to decrease the risk of cardiovascular events primarily by reducingSeptember 1, 2017: JAMA CardiologyAssociations of albuminuria in patients with chronic heart failure: findings in the ALiskiren Observation of heart Failure Treatment study.CONCLUSIONS: Increased UACR is common in patients with heart failure, including non-diabetics. Urinary albumin creatininineJul, 2011: European Journal of Heart FailureRandomized Controlled TrialEffects of Liraglutide on Clinical Stability Among Patients With Advanced Heart Failure and Reduced Ejection Fraction: A Randomized Clinical Trial.Review

Get seemless 1-tap access through your institution/university

For the best experience, use the Read mobile app

Read by QxMD is copyright © 2021 QxMD Software Inc. All rights reserved. By using this service, you agree to our terms of use and privacy policy.

You can now claim free CME credits for this literature searchClaim now

Get seemless 1-tap access through your institution/university

For the best experience, use the Read mobile app