ENGLISH ABSTRACT
JOURNAL ARTICLE
REVIEW
Add like
Add dislike
Add to saved papers

[Clinical aspect of recent progress in phosphate metabolism. Hyperphosphatemia and hypophosphatemia].

Serum inorganic phosphate (Pi) concentrations are generally maintained at 2.5-4.5 mg/dL in adults. In children, serum Pi levels are higher than those in adults. Hyperphosphatemia may be the consequence of (1) a decreased excretion of Pi, or (2) an external or internal cause of acute Pi loading to extracellular fluid, and the chronic kidney disease (CKD) is the most frequent cause of hyperphosphatemia. Hyperphosphatemia due to the increased Pi reabsorption is associated with hyperparathyroidism, tumoral calcinosis, and so on. Hypophosphatemia may be caused by (1) a decreased Pi intake, (2) an increased excretion of Pi, or (3) the transcellular shift of Pi from the extracellular to the intracellular space. Hypophosphatemia due to the increased excretion of Pi can be observed in various diseases, including Fanconi syndrome, hereditary hypophosphatemic rickets/osteomalacia, and tumor-induced hypophosphatemic osteomalacia.

Full text links

We have located links that may give you full text access.
Can't access the paper?
Try logging in through your university/institutional subscription. For a smoother one-click institutional access experience, please use our mobile app.

For the best experience, use the Read mobile app

Mobile app image

Get seemless 1-tap access through your institution/university

For the best experience, use the Read mobile app

All material on this website is protected by copyright, Copyright © 1994-2024 by WebMD LLC.
This website also contains material copyrighted by 3rd parties.

By using this service, you agree to our terms of use and privacy policy.

Your Privacy Choices Toggle icon

You can now claim free CME credits for this literature searchClaim now

Get seemless 1-tap access through your institution/university

For the best experience, use the Read mobile app