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Sophie J Clarke, Joel P Giblett, Lucy L Yang, Annette Hubsch, Tian Zhao, Muhammad Aetesam-Ur-Rahman, Nick E J West, Michael O'Sullivan, Nichola Figg, Martin Bennett, Nicolai J Wewer Albrechtsen, Carolyn F Deacon, Joseph Cheriyan, Stephen P Hoole
Background The mechanism underlying the beneficial cardiovascular effects of the incretin GLP-1 (glucagon-like peptide 1) and its analogues in humans is elusive. We hypothesized that activating receptors located on vascular smooth muscle cells to induce either peripheral or coronary vasodilatation mediates the cardiovascular effect of GLP -1. Methods and Results Ten stable patients with angina awaiting left anterior descending artery stenting underwent forearm blood flow measurement using forearm plethysmography and post-percutaneous coronary intervention coronary blood flow measurement using a pressure-flow wire before and after peripheral GLP -1 administration...
November 20, 2018: Journal of the American Heart Association
Muhammad Aetesam-Ur-Rahman, Colin Berry
No abstract text is available yet for this article.
June 2017: Cardiovascular Diagnosis and Therapy
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